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These potential therapies exert specific effects by targeting abnormal -syn aggregation, correcting brain iron dyshomeostasis, modulating cellular signaling pathways, among other mechanisms, and have demonstrated significant neuroprotective effects in preclinical studies (Table 2)

These are often in response to certain triggers, such as smoking or infection

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Buthionine sulfoximine (BSO), 1, 3-bis (2-chloroethyl)-1-nitrosourea (BCNU), diamide, and Hoechst 33342 were purchased from Sigma (St

In panels C and D, the reverse effect of N -acetyl cysteine in the presence of DMF/BSO treatment was assayed